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  • team
  • department
  • center
  • program_project
  • nrc
  • whocc
  • project
  • software
  • tool
  • patent
  • Administrative Staff
  • Assistant Professor
  • Associate Professor
  • Clinical Research Assistant
  • Clinical Research Nurse
  • Clinician Researcher
  • Department Manager
  • Dual-education Student
  • Full Professor
  • Honorary Professor
  • Lab assistant
  • Master Student
  • Non-permanent Researcher
  • Nursing Staff
  • Permanent Researcher
  • Pharmacist
  • PhD Student
  • Physician
  • Post-doc
  • Prize
  • Project Manager
  • Research Associate
  • Research Engineer
  • Retired scientist
  • Technician
  • Undergraduate Student
  • Veterinary
  • Visiting Scientist
  • Deputy Director of Center
  • Deputy Director of Department
  • Deputy Director of National Reference Center
  • Deputy Head of Facility
  • Director of Center
  • Director of Department
  • Director of Institute
  • Director of National Reference Center
  • Group Leader
  • Head of Facility
  • Head of Operations
  • Head of Structure
  • Honorary President of the Departement
  • Labex Coordinator
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About

Pedro Escoll received his B.Sc. in Biology from Universidad Autonoma de Madrid and obtained his Ph.D. in Immunology in the laboratory of Antonio de la Hera and Eva Sanz at Alcala University, Madrid, Spain, where he worked on TLR signaling and IL-1beta–Glucocorticoid receptor crosstalk.

In 2012, he joined the laboratory of Carmen Buchrieser at the Institut Pasteur Paris for post-doc training, and he investigated the influence of Legionella pneumophila infection over mitochondrial behavior.

Since 2018 he is permanent researcher at the “Biology of Intracellular Bacteria” (BBI) Unit, and his projects aim to understand how the immunometabolic interactions of human cells and intracellular bacteria impact infection.

He is currently the group leader of the “Metabolic host-bacteria interactions” team at the BBI Unit. His team uses Legionella pneumophila and Salmonella enterica serovar Thyphimurium as models to study the metabolic alterations induced by intracellular bacteria on human macrophages, and the impact of these metabolic shifts on the intracellular life of pathogenic bacteria during the course of infection.

Legionella pneumophila (green) interacts with mitochondria (red) during infection of a human macrophage

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