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© Research
Publication : Circulation

Protection against myocardial infarction and no-reflow through preservation of vascular integrity by angiopoietin-like 4

Team: Macrophages and Endothelial Cells
Member: Elisa Gomez Perdiguero
Scientific Fields
Diseases
Organisms
Applications
Technique

Published in Circulation - 15 Nov 2011

Galaup A, Gomez E, Souktani R, Durand M, Cazes A, Monnot C, Teillon J, Le Jan S, Bouleti C, Briois G, Philippe J, Pons S, Martin V, Assaly R, Bonnin P, Ratajczak P, Janin A, Thurston G, Valenzuela DM, Murphy AJ, Yancopoulos GD, Tissier R, Berdeaux A, Ghaleh B, Germain S

Link to Pubmed [PMID] – 22086875

Circulation 2012 Jan;125(1):140-9

Increased permeability, predominantly controlled by endothelial junction stability, is an early event in the deterioration of vascular integrity in ischemic disorders. Hemorrhage, edema, and inflammation are the main features of reperfusion injuries, as observed in acute myocardial infarction (AMI). Thus, preservation of vascular integrity is fundamental in ischemic heart disease. Angiopoietins are pivotal modulators of cell-cell junctions and vascular integrity. We hypothesized that hypoxic induction of angiopoietin-like protein 4 (ANGPTL4) might modulate vascular damage, infarct size, and no-reflow during AMI.