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© Research
Publication : Nature communications

Aquatic environment drives the emergence of cell wall-deficient dormant forms in Listeria.

Scientific Fields
Diseases
Organisms
Applications
Technique

Published in Nature communications - 02 Oct 2024

Carvalho F, Carreaux A, Sartori-Rupp A, Tachon S, Gazi AD, Courtin P, Nicolas P, Dubois-Brissonnet F, Barbotin A, Desgranges E, Bertrand M, Gloux K, Schouler C, Carballido-López R, Chapot-Chartier MP, Milohanic E, Bierne H, Pagliuso A

Link to Pubmed [PMID] – 39358320

Link to DOI – 10.1038/s41467-024-52633-7

Nat Commun 2024 Oct; 15(1): 8499

Stressed bacteria can enter a dormant viable but non-culturable (VBNC) state. VBNC pathogens pose an increased health risk as they are undetectable by growth-based techniques and can wake up back into a virulent state. Although widespread in bacteria, the mechanisms governing this phenotypic switch remain elusive. Here, we investigate the VBNC state transition in the human pathogen Listeria monocytogenes. We show that bacteria starved in mineral water become VBNC by converting into osmotically stable cell wall-deficient coccoid forms, a phenomenon that occurs in other Listeria species. We reveal the bacterial stress response regulator SigB and the autolysin NamA as major actors of VBNC state transition. We lastly show that VBNC Listeria revert to a walled and virulent state after passage in chicken embryos. Our study provides more detail on the VBNC state transition mechanisms, revealing wall-free bacteria naturally arising in aquatic environments as a potential survival strategy in hypoosmotic and oligotrophic conditions.