Ligne de temps

Sadri Znaidi
Chercheur(euse) Contractuel(le)
11 avr. 2024
publication

A gain-of-function mutation in zinc cluster transcription factor Rob1 Drives Candida albicans adaptive growth in the cystic fibrosis lung environment.

Lire plus
15 févr. 2023
project

Implementation of an NGS core facility at Institut Pasteur of Tunis: Biomics as capacity building model

Lire plus
01 janv. 2022
publication

Functional Portrait of Irf1 (Orf19.217), a Regulator of Morphogenesis and Iron Homeostasis in Candida albicans.

Lire plus
18 mars 2021
publication

Ethylzingerone, a Novel Compound with Antifungal Activity.

Lire plus
24 févr. 2021
publication

High Frequency of Enterocytozoon bieneusi Genotype WL12 Occurrence among Immunocompromised Patients with Intestinal Microsporidiosis.

Lire plus
04 déc. 2020
publication

A conserved regulator controls asexual sporulation in the fungal pathogen Candida albicans.

Lire plus
08 janv. 2020
publication

mSphere of Influence: Decoding Transcriptional Regulatory Networks To Illuminate the Mechanisms of Microbial Pathogenicity.

Lire plus
01 janv. 2020
publication

Identification and Characterization of Mediators of Fluconazole Tolerance in Candida albicans.

Lire plus
01 janv. 2019
publication

From Genes to Networks: The Regulatory Circuitry Controlling Candida albicans Morphogenesis.

Lire plus
01 déc. 2018
publication

A conserved fungal hub protein involved in adhesion and drug resistance in the human pathogen Candida albicans.

Lire plus
01 nov. 2018
publication

Systematic gene overexpression in Candida albicans identifies a regulator of early adaptation to the mammalian gut.

Lire plus
21 août 2018
publication

Generating genomic platforms to study Candida albicans pathogenesis.

Lire plus
28 juil. 2017
publication

The two-component response regulator Skn7 belongs to a network of transcription factors regulating morphogenesis in Candida albicans and independently limits morphogenesis-induced ROS accumulation.

Lire plus
29 mars 2016
publication

Mechanisms Underlying the Delayed Activation of the Cap1 Transcription Factor in Candida albicans following Combinatorial Oxidative and Cationic Stress Important for Phagocytic Potency.

Lire plus
02 sept. 2015
project

Candida albicans colonization of abiotic and biotic surfaces

Lire plus
02 sept. 2015
project

Regulation of Candida albicans morphogenesis

Lire plus
01 sept. 2015
project

FunComPath

Lire plus
01 sept. 2015
project

CANDIHUB

Lire plus
11 mai 2015
team

Biologie et Pathogénicité fongiques

Lire plus
11 déc. 2014
publication

Targeted changes of the cell wall proteome influence Candida albicans ability to form single- and multi-strain biofilms.

Lire plus
15 août 2013
publication

A comprehensive functional portrait of two heat shock factor-type transcriptional regulators involved in Candida albicans morphogenesis and virulence.

Lire plus
25 sept. 2012
publication

A versatile overexpression strategy in the pathogenic yeast Candida albicans: identification of regulators of morphogenesis and fitness.

Lire plus
01 janv. 2012
publication

A luciferase reporter for gene expression studies and dynamic imaging of superficial Candida albicans infections.

Lire plus
14 mars 2011
publication

Regulation of efflux pump expression and drug resistance by the transcription factors Mrr1, Upc2, and Cap1 in Candida albicans.

Lire plus
24 avr. 2009
publication

Identification of the Candida albicans Cap1p regulon.

Lire plus
04 avr. 2008
publication

Genomewide location analysis of Candida albicans Upc2p, a regulator of sterol metabolism and azole drug resistance.

Lire plus
28 sept. 2007
publication

Genome-wide expression and location analyses of the Candida albicans Tac1p regulon.

Lire plus
01 janv. 2007
publication

The zinc cluster transcription factor Tac1p regulates PDR16 expression in Candida albicans.

Lire plus
17 juin 2005
publication

A human-curated annotation of the Candida albicans genome.

Lire plus
10 déc. 2003
publication

The Schizosaccharomyces pombe corepressor Tup11 interacts with the iron-responsive transcription factor Fep1.

Lire plus