Timeline

01 Jul 2020

publication

Antiapoptotic Clone 11-Derived Peptides Induce In Vitro Death of CD4+ T Cells Susceptible to HIV-1 Infection.

01 Jul 2019

publication

Metabolic plasticity of HIV-specific CD8+ T cells is associated with enhanced antiviral potential and natural control of HIV-1 infection.

05 Mar 2019

publication

Cellular Metabolism Is a Major Determinant of HIV-1 Reservoir Seeding in CD4+ T Cells and Offers an Opportunity to Tackle Infection.

01 Jan 2018

publication

Cellular Determinants of HIV Persistence on Antiretroviral Therapy

11 Dec 2014

publication

BI-2 destabilizes HIV-1 cores during infection and Prevents Binding of CPSF6 to the HIV-1 Capsid

29 May 2014

publication

Effects of human SAMHD1 polymorphisms on HIV-1 susceptibility

12 Nov 2013

publication

Contribution of oligomerization to the anti-HIV-1 properties of SAMHD1

26 Apr 2013

publication

The ability of TNPO3-depleted cells to inhibit HIV-1 infection requires CPSF6

17 Apr 2013

publication

The retroviral restriction ability of SAMHD1, but not its deoxynucleotide triphosphohydrolase activity, is regulated by phosphorylation

13 Nov 2012

publication

Contribution of SAM and HD domains to retroviral restriction mediated by human SAMHD1

16 Oct 2012

publication

Contribution of SUMO-interacting motifs and SUMOylation to the antiretroviral properties of TRIM5α

17 Sep 2012

publication

Reduced connexin43 expression correlates with c-Src activation, proliferation, and glucose uptake in reactive astrocytes after an excitotoxic insult

12 Jun 2012

publication

Role of SAMHD1 nuclear localization in restriction of HIV-1 and SIVmac

23 Feb 2012

publication

HIF-1 and c-Src mediate increased glucose uptake induced by endothelin-1 and connexin43 in astrocytes

15 Jan 2009

publication

A little guide for advanced search:

Timeline

Antiapoptotic Clone 11-Derived Peptides Induce In Vitro Death of CD4+ T Cells Susceptible to HIV-1 Infection.

Metabolic plasticity of HIV-specific CD8+ T cells is associated with enhanced antiviral potential and natural control of HIV-1 infection.

Cellular Metabolism Is a Major Determinant of HIV-1 Reservoir Seeding in CD4+ T Cells and Offers an Opportunity to Tackle Infection.

Cellular Determinants of HIV Persistence on Antiretroviral Therapy

BI-2 destabilizes HIV-1 cores during infection and Prevents Binding of CPSF6 to the HIV-1 Capsid

Effects of human SAMHD1 polymorphisms on HIV-1 susceptibility

Contribution of oligomerization to the anti-HIV-1 properties of SAMHD1

The ability of TNPO3-depleted cells to inhibit HIV-1 infection requires CPSF6

The retroviral restriction ability of SAMHD1, but not its deoxynucleotide triphosphohydrolase activity, is regulated by phosphorylation

Contribution of SAM and HD domains to retroviral restriction mediated by human SAMHD1

Contribution of SUMO-interacting motifs and SUMOylation to the antiretroviral properties of TRIM5α

Reduced connexin43 expression correlates with c-Src activation, proliferation, and glucose uptake in reactive astrocytes after an excitotoxic insult

Role of SAMHD1 nuclear localization in restriction of HIV-1 and SIVmac

HIF-1 and c-Src mediate increased glucose uptake induced by endothelin-1 and connexin43 in astrocytes

Connexin43 is involved in the effect of endothelin-1 on astrocyte proliferation and glucose uptake