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© Research
Publication : The EMBO journal

NIX-ing mitochondria: from development to pathology.

Team: Brain-immune communication
Member: Aleksandra Deczkowska
Scientific Fields
Diseases
Organisms
Applications
Technique

Published in The EMBO journal - 14 Jun 2017

Deczkowska A, Schwartz M,

Link to Pubmed [PMID] – 28533231

Link to DOI – 10.15252/embj.201797110

EMBO J 2017 Jun; 36(12): 1650-1652

Hypoxia occurs physiologically in the developing body, and changing oxygen tensions are known to direct tissue differentiation; however, in the context of pathology, the same hypoxia‐activated mechanisms may negatively affect tissue function. In this issue of The EMBO Journal, Esteban‐Martínez et al (2017) report that programmed mitophagy, dependent on hypoxia‐induced NIP‐3‐like protein X (BNIP3L, best known as NIX), is an essential step in differentiation of both retinal neurons and inflammatory macrophages.