About
Epidemiological studies show that genital infections with the intracellular bacteria Chlamydia trachomatis increase the risk of cervical cancer, but the underlying mechanisms are still elusive. C. trachomatis infection causes DNA damage in the host cells, which could favor cellular transformation if these cells survive the infection and multiply. This project aims at exploring, at a deep mechanistic level, the causal link between C. trachomatis and DNA damage in the host, and at investigating the long-term consequences of the damages.
