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Exploitation of host cell cytoskeleton and signalling during Listeria monocytogenes entry into mammalian cells

Adjuvant activity of Mycobacterium bovis BCG expressing CRM197 on the immune response induced by BCG expressing tetanus toxin fragment C

© Charles Dauguet

Virus VIH-1 (HIV-1), agent du sida, à la surface d'un lymphocyte. Image colorisée.

Publication : Arteriosclerosis, thrombosis, and vascular biology

Human apolipoprotein A-IV reduces secretion of proinflammatory cytokines and atherosclerotic effects of a chronic infection mimicked by lipopolysaccharide

Team: Archived: Group: Daniel Scott-Algara

Team: Archived: Regulation of Retroviral Infections

Member: Daniel Scott-Algara

Scientific Fields

Diseases

Organisms

Applications

Technique

Published in Arteriosclerosis, thrombosis, and vascular biology - 29 Jan 2004

Recalde D, Ostos MA, Badell E, Garcia-Otin AL, Pidoux J, Castro G, Zakin MM, Scott-Algara D

Link to Pubmed [PMID] – 14751811

Arterioscler. Thromb. Vasc. Biol. 2004 Apr;24(4):756-61

Expression of human apolipoprotein (h-apo) A-IV in apoE-deficient (apoE(0)) mice (h-apoA-IV/E(0)) reduces susceptibility to atherosclerosis. Chronic infection mimicked by exposure to lipopolysaccharide (LPS) increases the size of atherosclerosis lesions in apoE(0) mice. Thus, we used h-apoA-IV/E(0) mice to determine whether h-apoA-IV plays a protective role after LPS administration.

Published on: 29 Jan 2004