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© Charles Dauguet
Virus VIH-1 (HIV-1), agent du sida, à la surface d'un lymphocyte. Image colorisée.
Publication : Arteriosclerosis, thrombosis, and vascular biology

Human apolipoprotein A-IV reduces secretion of proinflammatory cytokines and atherosclerotic effects of a chronic infection mimicked by lipopolysaccharide

Scientific Fields
Diseases
Organisms
Applications
Technique

Published in Arteriosclerosis, thrombosis, and vascular biology - 29 Jan 2004

Recalde D, Ostos MA, Badell E, Garcia-Otin AL, Pidoux J, Castro G, Zakin MM, Scott-Algara D

Link to Pubmed [PMID] – 14751811

Arterioscler. Thromb. Vasc. Biol. 2004 Apr;24(4):756-61

Expression of human apolipoprotein (h-apo) A-IV in apoE-deficient (apoE(0)) mice (h-apoA-IV/E(0)) reduces susceptibility to atherosclerosis. Chronic infection mimicked by exposure to lipopolysaccharide (LPS) increases the size of atherosclerosis lesions in apoE(0) mice. Thus, we used h-apoA-IV/E(0) mice to determine whether h-apoA-IV plays a protective role after LPS administration.

http://www.ncbi.nlm.nih.gov/pubmed/14751811