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Veterinary
Visiting Scientist

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Deputy Director of Department
Deputy Director of National Reference Center
Deputy Head of Facility
Director of Center
Director of Department
Director of Institute
Director of National Reference Center
Group Leader
Head of Facility
Head of Operations
Head of Structure
Honorary President of the Departement
Labex Coordinator

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Virus entry: old viruses, new receptors.

RANKL induces organized lymph node growth by stromal cell proliferation

Publication : Journal of pediatric endocrinology & metabolism : JPEM

Duplication of SOX9 is not a common cause of 46,XX testicular or 46,XX ovotesticular DSD

Team: Human Developmental Genetics

Member: Kenneth McElreavey

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Diseases

Organisms

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Published in Journal of pediatric endocrinology & metabolism : JPEM - 01 Jan 2012

Seeherunvong T, Ukarapong S, McElreavey K, Berkovitz GD, Perera EM

Link to Pubmed [PMID] – 22570960

J. Pediatr. Endocrinol. Metab. 2012;25(1-2):121-3

Translocation of the SRY gene to the paternal X chromosome is the explanation for testis development in the majority of subjects with 46,XX testicular disorder of sexual development (DSD). However, nearly all subjects with 46,XX ovotesticular DSD and up to one third of subjects with 46,XX testicular DSD lack SRY. SRY-independent expression of SOX9 has been implicated in the etiology of testis development in some individuals.

Published on: 01 Jan 2012