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2024Measuring single-cell susceptibility to antibiotics within monoclonal bacterial populations, https://doi.org/10.1371/journal.pone.0303630.
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2024Aminoglycoside tolerance in Vibrio cholerae engages translational reprogramming associated to queuosine tRNA modification, Elife.
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2022Systematic transcriptome analysis allows the identification of new type I and type II Toxin/Antitoxin systems located in the superintegron of Vibrio cholerae., Res Microbiol 2023 ; 174(1-2): 103997.
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2022Nonessential tRNA and rRNA modifications impact the bacterial response to sub-MIC antibiotic stress, microLife, Volume 3, 2022, uqac019.
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2022A qnr-plasmid allows aminoglycosides to induce SOS in Escherichia coli., Elife 2022 Jan; 11(): .
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2021Interplay between Sublethal Aminoglycosides and Quorum Sensing: Consequences on Survival in V. cholerae., Cells 2021 11; 10(11): .
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2021Sleeping ribosomes: Bacterial signaling triggers RaiA mediated persistence to aminoglycosides., iScience 2021 Oct; 24(10): 103128.
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2021Deficiency in cytosine DNA methylation leads to high chaperonin expression and tolerance to aminoglycosides in Vibrio cholerae., PLoS Genet 2021 10; 17(10): e1009748.
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2019RadD Contributes to R-Loop Avoidance in Sub-MIC Tobramycin., mBio 2019 07; 10(4): .
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2014SOS, the formidable strategy of bacteria against aggressions., FEMS Microbiol Rev 2014 Nov; 38(6): 1126-45.
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2014Influence of very short patch mismatch repair on SOS inducing lesions after aminoglycoside treatment in Escherichia coli, Res. Microbiol. 2014 Jul-Aug;165(6):476-80.
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2014Identification of genes involved in low aminoglycoside-induced SOS response in Vibrio cholerae: a role for transcription stalling and Mfd helicase., Nucleic Acids Res 2014 Feb; 42(4): 2366-79.
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2013Multiple Pathways of Genome Plasticity Leading to Development of Antibiotic Resistance., Antibiotics (Basel) 2013 May; 2(2): 288-315.
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2013RpoS plays a central role in the SOS induction by sub-lethal aminoglycoside concentrations in Vibrio cholerae., PLoS Genet 2013 ; 9(4): e1003421.
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2013β-Lactam antibiotics promote bacterial mutagenesis via an RpoS-mediated reduction in replication fidelity, Nat Commun 2013;4:1610.
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2012Connecting environment and genome plasticity in the characterization of transformation-induced SOS regulation and carbon catabolite control of the Vibrio cholerae integron integrase., J Bacteriol 2012 Apr; 194(7): 1659-67.
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2011Vibrio cholerae triggers SOS and mutagenesis in response to a wide range of antibiotics: a route towards multiresistance., Antimicrob Agents Chemother 2011 May; 55(5): 2438-41.
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2011Formation of a stable RuvA protein double tetramer is required for efficient branch migration in vitro and for replication fork reversal in vivo, J. Biol. Chem. 2011 Jun;286(25):22372-83.
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2010Folded DNA in action: hairpin formation and biological functions in prokaryotes., Microbiol Mol Biol Rev 2010 Dec; 74(4): 570-88.
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2010Conjugative DNA transfer induces the bacterial SOS response and promotes antibiotic resistance development through integron activation., PLoS Genet 2010 Oct; 6(10): e1001165.
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2010RNA polymerase mutations that facilitate replication progression in the rep uvrD recF mutant lacking two accessory replicative helicases, Mol. Microbiol. 2010 Jul;77(2):324-36.
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2008ruvA and ruvB mutants specifically impaired for replication fork reversal, Mol. Microbiol. 2008 Oct;70(2):537-48.
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2008ruvA Mutants that resolve Holliday junctions but do not reverse replication forks, PLoS Genet. 2008 Mar;4(3):e1000012.
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2007Recombination proteins and rescue of arrested replication forks, DNA Repair (Amst.) 2007 Jul;6(7):967-80.
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2006RuvAB is essential for replication forks reversal in certain replication mutants, EMBO J. 2006 Feb;25(3):596-604.