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© Artur Scherf
Scanning Electron Microscopy of Red Blood Cell infected by Plasmodium falciparum.
Publication : Annales de dermatologie et de vénéréologie

[Immunological and non immunological mechanisms in urticaria]

Scientific Fields
Diseases
Organisms
Applications
Technique

Published in Annales de dermatologie et de vénéréologie - 01 May 2003

Bérard F, Saint-Mezard P, Cousin F, Mecheri S, Nicolas JF

Link to Pubmed [PMID] – 12843804

Ann Dermatol Venereol 2003 May;130 Spec No 1:1S10-5

Urticaria involve mast cell activation which could be mediated by immunological or non-immunological mechanisms. Interaction of allergens with the IgE/IgE receptor at the surface of mast cells has been postulated as the main immunologic type of mast cell activation. However, recent experimental and clinical studies have highlighted the existence of other mechanisms involving specific antibodies and T cells. IgG antibodies of different specificities (anti-IgE and/or anti-IgE receptor autoantibodies) have been characterized in a subgroup of patients suffering from chronic “autoimmune” urticaria. Circulating immune complexes may activate mast cells by interaction with the membrane-bound receptor for IgG. Interaction of mast cells with specific T cells could induce mast cell activation. Thus, immune-mediated urticaria appears to be secondary to different types of mast cell activation which could explain the various clinical presentation of the disease.

http://www.ncbi.nlm.nih.gov/pubmed/12843804