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Genome stability in quiescence
Benoit Arcangioli (PI)
In nature, cells alternate between replicative and non-replicative states depending on the fluctuating environmental conditions and physiological requirements (Koch, 1971). The non-replicative state resulting from cell division arrest or transition to a more specialized […]
2022The quiescent X, the replicative Y and the Autosomes, Peer Community Journal, 2: e18.
2020Nitrogen starvation reveals the mitotic potential of mutants in the S/MAPK pathways., Nat Commun 2020 04; 11(1): 1973.
2018Molecular signature of the imprintosome complex at the mating-type locus in fission yeast, Microb Cell 2018 Jan;5(4):169-183.
2017Quiescence unveils a novel mutational force in fission yeast, Elife 2017 Dec;6.
2017DNA Repair and Mutations During Quiescence in Yeast, FEMS Yeast Res. 2017 Jan;.
2013Srs2 mediates PCNA-SUMO-dependent inhibition of DNA repair synthesis, EMBO J. 2013 Mar;32(5):742-55.
2010Stable interactions between DNA polymerase δ catalytic and structural subunits are essential for efficient DNA repair, DNA Repair (Amst.) 2010 Oct;9(10):1098-111.
2010The RecQ DNA helicases in DNA repair, Annu. Rev. Genet. 2010;44:393-417.
2008Srs2 removes deadly recombination intermediates independently of its interaction with SUMO-modified PCNA, Nucleic Acids Res. 2008 Sep;36(15):4964-74.
2008The Srs2 helicase activity is stimulated by Rad51 filaments on dsDNA: implications for crossover incidence during mitotic recombination, Mol. Cell 2008 Feb;29(2):243-54.
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