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© A-M. Pais-Correia, M-I. Thoulouze, A. Alcover, A. Gessain
Mise en évidence de structures de type "biofilm ", formées par le rétrovirus HTLV-1 générés par des cellules infectées (cellules du haut), qui ont été transmis à un autre lymphocyte (cellule du bas). Micrographie en microscopie électronique à balayage. Image colorisée.
Publication : Retrovirology

ADAR1 enhances HTLV-1 and HTLV-2 replication through inhibition of PKR activity

Team: Oncogenic Virus Epidemiology and Pathophysiology
Team: Archived: Group: Jean-Pierre Vartanian
Team: Archived: Molecular Retrovirology
Scientific Fields
Diseases
Organisms
Applications
Technique

Published in Retrovirology - 12 Nov 2014

Cachat A, Alais S, Chevalier SA, Journo C, Fusil F, Dutartre H, Boniface A, Ko NL, Gessain A, Cosset FL, Suspène R, Vartanian JP, Mahieux R

Link to Pubmed [PMID] – 25389016

Retrovirology 2014;11:93

The role of innate immunity in general and of type I interferon (IFN-I) in particular in HTLV-1 pathogenesis is still a matter of debate. ADAR1-p150 is an Interferon Stimulated Gene (ISG) induced by IFN-I that can edit viral RNAs. We therefore investigated whether it could play the role of an anti-HTLV factor.