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Deputy Director of Department
Deputy Director of National Reference Center
Deputy Head of Facility
Director of Center
Director of Department
Director of Institute
Director of National Reference Center
Group Leader
Head of Facility
Head of Operations
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Honorary President of the Departement
Labex Coordinator

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Inducible expression, enzymatic activity, and origin of higher plant homologues of bacterial RelA/SpoT stress proteins in Nicotiana tabacum

Dose effect relationship between the number of normal progenitor muscle cells grafted in mdx mouse skeletal striated muscle and the number of dystrophin-positive fibres

© Institut Pasteur

Culture de myotubes murins infectés par le virus de la rage fixe, observée en immunoflorescence indirecte.

Publication : Annals of the New York Academy of Sciences

Glycoprotein of nonpathogenic rabies viruses is a major inducer of apoptosis in human jurkat T cells

Team: Viral Neuro-Immunology

Member: Monique Lafon

Scientific Fields

Diseases

Organisms

Applications

Technique

Published in Annals of the New York Academy of Sciences - 01 Dec 2003

Lay S, Préhaud C, Dietzschold B, Lafon M

Link to Pubmed [PMID] – 15033795

Ann. N. Y. Acad. Sci. 2003 Dec;1010:577-81

This study sought to identify the RV protein that causes apoptosis. For this purpose, we first compared the ability of G and N proteins of a pathogenic and a nonpathogenic strain to trigger apoptosis of Jurkat rtTA by using an inducible Tet-on expression system. Then we analyzed apoptosis induced by a reverse genetic-engineered recombinant rabies virus in which the G gene from a nonpathogenic strain was replaced by its pathogenic strain counterpart. No other virus proteins than G of nonpathogenic RV strains induce apoptosis, and the G polypeptide of RV is a critical determinant for apoptosis in human cells.

Published on: 01 Dec 2003