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© Valérie Choumet
Mosquitoes were orally infected with the chikungunya virus. Midguts were dissected at day 5 post-infection, fixed and permeabilised. Virus is shown in red (anti-E2 protein, cyanine 3), the actin network in green (phalloidin 548) and nuclei in blue (DAPI).
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About

I completed my undergraduate, MSc and PhD degrees at the University of the Witwatersrand in Johannesburg, South Africa. From early on in my career, I was interested in biological control of insects – a subject that eventually brought me to research on mosquitoes, and in particular, vector control of Anopheles species. I worked largely on assessing underlying genetic mechanisms associated with insecticide resistance in Anopheles arabiensis during my PhD, and later on wild populations of An. gambiae and An. funestus from the Democratic Republic of Congo – a country marred by political and socio-economic problems and hence under-studied in this regard, despite the severe burden of malaria in the country. I joined Ken Vernick’s group in June of 2015 and have expanded my research interests to understanding the evolutionary mechanisms driving speciation between An. gambiae and An. coluzzii, two of the most important African malaria vectors. Further, I have maintained my interest in insecticide resistance and through the use of functional genomics, am trying to improve our understanding of target-site mutations and their mechanisms in conferring resistance, as well as the implications of their presence for host physiology and vectorial capacity.

Outside of work, I enjoy Ultimate frisbee, running, yoga, hiking, travel, reading, knitting, good food, good wine and seeing friends.

CV

https://www.researchgate.net/profile/Luisa_Nardini